My understanding is that the L form bacteria mutates the gene/s over a period of time, so if for example someone ends up with rhumatoid arthritis as a result of L form bacteria which has mutated a gene and 2 generations later a family member contracts rhumatoid arthritis due to inherited mutated gene, then how are they able to be treated with MP if they only have a mutated gene and not the bacteria. Or have i misunderstood the whole mutated gene "thing"????
It's turning out that 'disease genes' are largely a red herring. There are genetic markers that are associated with diseases, but the correlations are often quite weak. This is because what is actually being observed is genetic damage in infected cells.
My understanding is that it's actually pretty rare for the damaged genes to be passed on to the next generation, but the L-forms are definitely transferred between family members. So similar damage will show up in the descendants because they are infected too.
When the immune system is working properly, the infected cells are destroyed, thus eliminating the damaged DNA.
____________________ 5/6 CFS criteria, 125D36 Ph1Jul08 Ph2Sep08 25D8(Sep08) NoIRs covered up (except hands) low lux home minimal light exp r/t work
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